What is cracked tooth syndrome?
Syndrome Capgras
Cracked tooth syndrome is difficult to diagnose because the fracture is usually too small to be detected with an X-ray. Gross will inspect your teeth for cracks and ask you to bite on a plastic tool that covers one tooth at a time to determine which tooth is experiencing pain. When the cracked tooth syndrome diagnosis is confi rmed, an initial occlusal adjustment should be made to remove occlusal interferences and relieve symptoms (7), and then a restoration should be. Cracked tooth syndrome may be defined as a tooth fracture plane of unknown depth, which originate from the crown, passes through the tooth structure involving the dentine and occasionally extends into the pulp and extends subgingivally, and may progress to connect with the pulp space and/or periodontal ligament.
The enamel that makes up our teeth is the hardest substance in our body. It’s even harder than bone. But the wear and tear we place on our teeth through biting, grinding, and trauma can still cause cracks in our teeth. Sometimes the cracks are so small that you can’t even see them, but they’ll still cause the pain and discomfort of cracked tooth syndrome (CTS).
These are the major symptoms for CTS:
- pain when biting, especially when the bite is released
- pain when eating or drinking cold or sugary foods or beverages
- Although any tooth can crack, cracks usually occur to the back molars. These teeth get a lot of use every day and can get a further workout if you grind or clench your teeth.
- Cracks can be small and only effect the outer tooth enamel. They might cause no pain or they can extend through the tooth and down into the root of the tooth and cause severe pain.
- A small crack, if not treated, can lead to a lengthening of the crack and the symptoms of CTS, including infection and pain.
How to diagnose cracked tooth syndrome
Cracked tooth syndrome is one of the most difficult disorders to diagnose. Often the crack in the tooth is not visible, even in an x-ray. Dentists have special tools, like microscopes, dyes, and ‘bite tests’ to find a crack in a tooth and get to the root of the cause.
How to treat cracked tooth syndrome
Cracked tooth repair depends on the extent of the crack, the location of the crack and the health of the effected tooth. Cracks in teeth, unlike cracks in bones, do not heal. The goal of treatment is to prevent worsening of the crack and to save the tooth. https://beachkehiwi1989.wixsite.com/softmac/post/firefox-34-0.
Possible treatments include:
Syndrome Rachidienne
- Root canal therapy
- There are instances when extraction of the tooth may be necessary, so it’s best to try to prevent cracks in teeth.
How can cracked tooth syndrome be prevented?
![Crack Crack](/uploads/1/2/8/3/128395748/863229605.jpg)
Because we are always biting and chewing, it’s not always possible to completely prevent cracks in teeth. But there are some things you can do to reduce strain on your teeth, like:
- not clenching or grinding your teeth
- not chewing on hard objects, like ice
- wearing a mouthguard at night if you grind your teeth while you sleep
- wearing a face guard or mouth guard while paying contact sports
Get a checkup
To sum up, getting regular dental care is the best way to keep your teeth healthy and clean and prevent CTS. So, contact us for an appointment today.
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With the arrival of the relatively inexpensive and smokable crack form of cocaine, the rate of newborns exposed to cocaine in utero rose dramatically in the early 1990s. In the mid-1990s it was estimated that 1.1% of pregnant women used crack cocaine. Early reports of neonatal behavioral abnormalities led to the label “crack baby syndrome” and were followed up with several large, longitudinal studies. Screen rotation windows 10. Crack cocaine–exposed neonates were demonstrated to be more jittery and less likely to modify their attentional behavior based on their arousal state. During the first year, infants exposed prenatally to crack cocaine are rated as having more attentional abnormalities and being less successful at remembering stimuli in their environments. However, the term crack baby syndrome is now generally recognized as inappropriate beyond the early neonatal period, after which time the behavioral differences are more subtle and do not constitute a distinct syndrome.
Several large-scale studies were funded to follow crack cocaine–exposed babies over time. They found that the mothers who had used crack cocaine during pregnancy were different from pregnant women recruited from similar economic and ethnic/racial groups. The mothers of the cocaine-exposed infants were (1) more likely to have used alcohol, cigarettes, and marijuana during pregnancy; (2) were more likely to continue to use alcohol and drugs of abuse postnatally; and (3) were more likely to have symptoms of psychiatric distress prenatally/postnatally. These polysubstance use and psychological characteristics of crack cocaine–using women have been repeatedly documented and found to have their own negative effects on child development.
One question addressed by these studies is to what degree are any persistent difficulties seen due to the cocaine exposure itself, not the other risk factors. In regards to overall mental functioning in early childhood, some studies found small effects of the cocaine specifically and others found no effect. However, specific effects of cocaine exposure on global functioning reported in infancy were no longer detectable, or subtle, by school age.
However, several studies have shown that cocaine exposure is associated with disruptions in attention and inhibition that could not be attributed to differences in maternal functioning and exposure to other substances. These independent relations of cocaine exposure on attention and inhibition have been confirmed in animals and persist into school age. In particular, the difficulties seem to be in paying attention to the information that is important in the moment and using that information to reach goals, as opposed to acting impulsively. Unfortunately, the attentional and planning abilities that are particularly sensitive to crack cocaine are also vulnerable to alcohol, cigarette, and marijuana use during pregnancy. So although the specific effect of the cocaine may be small, crack cocaine–exposed children usually are exposed to multiple substances that all impact on the same system.
Although it may not ameliorate the effects of cocaine on attention, positive environments have been shown to reduce the rate of mental retardation and increase verbal IQ in cocaine-exposed children. The hopeful findings of researchers is that providing a home life supportive of mental development makes a substantial impact on how crack cocaine–exposed children are doing as they enter school age.
Crack Tooth Syndrome
References:
Syndrome Crouzon
- Bendersky, , Gambini, G., Lastella, A., Bennet, D. S., & Lewis, M. (2003). Inhibitory motor control at five years as a function of prenatal cocaine exposure. Journal of Developmental Behavioral Pediatrics, 24, 345–351.
- Harvey, A., & Kosofsky, J. A. (Eds.). (1998). Cocaine: Effects of the developing brain. Annals of the New York Academy of Sciences, 846.
- National Clearinghouse for Drug and Alcohol Abuse Information, http://www.health.org
- National Institute of Drug Abuse of the National Institute of Health, http://www.drugabgov
- Singer, T., Minnes, S., Short, E., Arendt, R., Farkas, K., Lewis, B., et al. (2004). Cognitive outcomes of preschool children with prenatal cocaine exposure. Journal of the American Medical Association, 291, 2448–2456.